The Skull Trap

The Skull Trap
Andre Willers
22 Jan 2012

Synopsis:
The skull is a poor place to put a brain . Eating and thinking makes for a rather limited fellowship .

Discussion :
1.The brain development was driven by one major factor : the amygdala .
Fast recognition and reaction to threats overrode everything else .
Other factors were :
2.Optic systems
3.Ganglia for movement
4.Memory of rewards .

A false optimum in evolution linked the four factors above to be in close proximity .
The Brain in the Skull .

This split happened about 300 MYA .
Amphibians' “brains” were distributed ,
Dinosaurs and mammals also have distributed brains , but to a much lesser extent .
See “A brief history of the Brain” , NewScientist 24 sep 2011 p 40
This led to humans having a severely limited processing capacity , due to skull size .

I am more interested in enhancing human capability .

Multiple Amygdala's
Humans already have this in simple forms. Also known as chakra's .
See Appendix I for Lower Back Pain . A local amygdala effect .

Creation and steering of local amygdala's using APS .
This should drastically increase sporting performance .

But will it make you smarter ?
The answer is yes , to a limited extent .

Shifting decision processing to local amygdala's frees the central brain from these tasks .
There is a leverage effect in that many nodes in the Mirror-Neuron Networks are freed up as well .

Super Sportsman .
This will give you very fast reflexes . Also more stamina (some housekeeping muscle systems are controlled from these ganglia) .

Genetic :
Implanting or stimulating amygdala stem cells in the chakra points should have some very interesting results .

Controlled cancers stimulated by amygdala's will result in brain mass in the body , where it belongs .

Will this trigger some epigenetic responses ? I have no idea , but suspect that some “fast-forward” cancer biolabs in the body already has some pre-prepared genes .

Drugs :
Identifying chakra's as amygdala's opens the whole toolbox of interventions ..
Propanolol an Imipramine springs to mind .

Take a lot of fish oil (omega3 fats . Etc)

Another fishy story .

Andre .

Ps. Correlate Fleming Effect with Caeserians .

Xxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxx
Appendix I

Peripheral Neuropathy of Legs. A Cure.
Andre Willers
20 Apr 2010
 
My Birthday Present to fellow sufferers .
 
Synopsis:
A methodology for reducing chronic symptoms (95%)  and repairing nerves .
 
Summary:
Data from my personal experience .
1.Use APS as for lower back pain once a day (8 min) . Unease relief 80%-85% .
2.Use capsaicum on upper thighs . Before sleep . Unease relief 10% . The need for this will decrease after about 5-7 usages of APS usage .
3. Keep blood-glucose concentration <=6.5
4. Do not eat high acrylamide foods
5.Take alpha-lipoic acid . 250 mg (1+2+1)  per day initially .Taper .
Beware reflux . An amusing side effect . Rejection reflexes of crap food are repaired .
Not beloved by the fast-food industry .
(Pregnant women should not take this in large doses. )
 
Discussion :
The pathology is caused by damage to the nerves , (especially the long leg-nerves) by high glucose concentrations in DiabetesII and Acrylamide from diet .
 
How it works:
The pain-pulses of damaged nerve A arrive at the synapses in the lower back (spine) . Here , certain specialized glial cells count the frequency and duration . To prevent the gate-effect of pain impulses to mask further pain signals of further tissue damage from other nerves , these cells take on the function of automatically pulsing pain signals as if from nerve A , while inhibiting the damaged nerve A's signal .
 
This enables other damage signals to come through , preventing further damage .
This has obvious evolutionary advantages in preventing cascading damages .
 
The problem is that , even if damage to nerve A has been repaired , the off-switch of this mechanism may not work . Or the damage to nerve A might remain .
 
See http://andreswhy.blogspot.com "Peripheral Neuropathy" et al .
Chronic neuropathy results from the inability to activate the "Off" switch in microglial and astrocyte bodies in the dorsal root ganglion .
See Scientific American Nov 2009 p 33 .
 
A further teensy problem is that , even if there was a temporary repair , if the chronic condition (like too high glucose or acrylamide concentrations) the microglial and astrocyte bodies get re-triggered . And they would be sensitized as well .
 
 
The Methodology :
1.Drastically reduce the pain impulses from the microglial and astrocyte bodies in the dorsal root ganglion . For this we use the APS technology , with electrodes configured for "Back Pain" .
See www.apstherapy.com
See http://andreswhy.blogspot.com "ATP as Neurotransmitter"
 
I originally bought this machine in 2001 for Neuropathy leg pains , but put the electrodes on the legs . This had little or no effect . Only after reading the article :
See Scientific American Nov 2009 p 33  did I realize that the chronic pain is generated in the lower back .
Putting the electrodes on the sides of the lower hip as recommended for lower back pain led to a reduction of that chronic burning unease sensation of 80-85 percent.
After about 4-7 days .
 
Some of the remainder of unease could be ameliorated by using gate-theory : capsaicum . Just smear it on the thighs . This generates pain signals that compete at the lowest spine ganglia , but unfortunately the system habituates easily . .
 
This brings us to about 95% of unease .
 
At least I could sleep without analgesics .
 
But there are still twinges .
 
The damage to the nerves still exist . The neuropathic pain will quickly re-establish itself . We are just managing the condition , not curing it .
 
The Cure :
Repair the nerves , while keeping the damaging agents under control .
The damaging agents are too high concentrations of glucose and acrylamide .
Keep them under control via diet or insulin .
 
The repair is enhanced by alpha-lipoic acid (evening primrose oil).
Take supplements .
 
Desensitizing the system :
A really permanent , buffered system . I do not have that .
Working ,working ….
 
A promising lead is propranolol . Reprogramming memories at synapse level (ie the microglial and astrocyte bodies in the dorsal root ganglion .)
Google "propranolol PTSD"
 
Happy Birthday !
 
Andre