Wednesday, May 02, 2012

Hold your breath .

Hold your breath
Andre Willers
2 May 2012
“And in this harsh world draw thy breath in pain ” . Shakespeare
Breathing has a primary pacemaker associated with microglial and astrocyte bodies in the root ganglions of the spine near the diaphragm . This can be modified by input from the brain

Please don’t try anything discussed here without qualified supervision .

1.The abdominal diaphragm controls breathing , especially the BreakPoint , where inhalation becomes involuntary . See “ScientificAmerican” Apr 2012 p65 “The limits of breath holding”

2.There is a Primary Pacemaker for breathing (12/min for humans) that directly times the diaphragm contraction . This is associated with microglial and astrocyte bodies in the root ganglions of the spine near the diaphragm . See Appendix I below for a neuropathy with a similar system . This pacemaker is designated as a chakra by Eastern systems .

3.From evolutionary reasons , the Breathing System would have evolved first , and was later adapted to monitor repetitive stress or pain systems and replace them , freeing up the primary nervous system . It was evolved to take enormous damage and still keep functioning .

4.It acts like a subsidiary amygdala , with it’s own memory of threats and responses . Anti-depressants (especially in the mother) would seem to be contra-indicated until more is known

It would be sensitive to the same factors causing Peripheral Neuropathy , especially in infants . Acrylamide should be avoided . (A possible cause of Sudden Infant Death Syndrome , where the infant just stops breathing) .

6.Now , How to Hold Your Breath .
6.1The system will not allow overriding of Reserves (1/3) , as discussed previously ad nauseam .
6.2Increase reserves by lowering metabolism . MSM , followed by a whiff of H2S should decrease metabolic rates (see Hibernation posts) , and prepare these very old microglial and astrocyte systems for a drastic slowdown (amygdala lessening-of-threat reponse)
6.3Some beta-blockers , especially propranolol would have a synergistic effect .
6.4A fairly large dose of fast melatonin (6 to 9 mg) would pulse-release orexins , leading to a drastic metabolic slowdown , affecting the estimate of oxygen reserve needed .
6.5Capsaicums : rubbing capsaicums into the vertebra at the critical spinal nerve junctions (chakra) will amount to what we would call a “denial of service” attack . The pacemaker still functions , but the signal is only transmitted say 1 in2 or 1 in 3 times normal . A severe slowdown (This is why capsaicums evolved as a poison)
6.6Alcohol : This will suppress breathing amygdala threat response in moderate doses , but too much and you will stop breathing (Sudden Stupid Infant Death Syndrome-SSIDS)
6.7Electronic means
See and posts on neuropathy and diabetes .
It works for Diabetic Neuropathy (see posts) , but monkeying around the breathing chakra requires some care .
6.8 !Click
Click languages can play a role . See Chi systems and Click posts .

The aim here is to enable you to hold your breath for longer without excessive sense of strain or exercise of willpower .
Why ?
Sports , of course .
See Prof Tim Noakes on the 1/3 reserve withholding . Or just sprinting faster (anaerobic) . A human should be able to run the 100 m in about 6 seconds using techniques like this .
Or swim 1/3 faster . Or just last longer .
Will human systems evolve with this ? Look for very specific prions in East Africa (long distance cursorial raptors – pay attention to !click ditties).

Supervision Required !
These techniques (or combinations of them) actually work , but a permanent state of holding-your-breath might result . Please use qualified supervision .

Each breath should be a pleasure .
Appendix I
Peripheral Neuropathy .
Andre Willers
18 Dec 2009

Synopsis :
Chronic neuropathy results from the inability to activate the "Off" switch in microglial and astrocyte bodies in the dorsal root ganglion .
See Scientific American Nov 2009 p 33 .

Discussion .

These bodies monitor the frequencies of pulses of neurotransmitters from the neuron . If there is neural damage (like with acrylamide cf) , they detect this and increase the sensitivity of the neurons , while also releasing cytokines and some interleukins for repairs . (ie inflammatory response .)

The "Off"-switch for these systems could do with some redesign .
The off-switch is related to the percussive frequency of the heartbeat . And this has harmonics with the length of the spine . And this changes with posture .

The percussion changes the densities of fluid inside the dorsal ganglion , influencing the pulses of neurotransmitters . Note that microglia are responsible for re-uptake of neurotransmitters . Cf prozac. Cf yoga .

If they kick in , fewer neurotransmitters are recycled . Boosting the signal .

If the heartbeat returns to normal , fine .

But , stress .

The kicker is the length of the spine .

This changes the resonances inside the dorsal root ganglion . The microglia and astrocytes do not switch off . Neural sensitivity is enhanced , and typical peripheral neuropathy symptoms ensue .
Like when you lie down to sleep .

What to do .

1."Put a bolster under the bum , mum ."
Compress the spine mildly by putting a cushion under the bum , while having high cushions for the upper torso . This enables the off-switch .
This actually works . (like sitting in small gravity)

Percussions , either by mouth , or mechanical , can modify synapse responses .
Cf all those relaxing massages .
Some skill needed .

3.Retrain the system .
Even more skill needed .

A note on the Gate-theory of Pain .
Using something like capsaicum is a very short-term solution . From the argument above , the system will habituate as the microglials and astrocytes compensate for the storm of incoming signals .
Ditto for more powerful pain stillers .

Some intriguing speculations:
1.Phantom limb pain .
Try compressing the limb in imagination . The chronic pain switches should then at least have a chance of switching off .
Do it even with existing limbs .

2.Alcohol .
The energy systems powering the microglials have the same imperatives as the rest of the mitochondria : alcohol has to be metabolized first .
A prolonged pulse of alcohol then causes an absence of pain . The pulses simply do not happen , and upstream consequences in the amygdala does not occur .

This causes an interrupt/reset in the microglial system . Which is why it so popular with humans .

The microglial system is the memory of pain . A little bit is good , but too much leads to learned helplessness and catatonia .
And the off-switch is interrupted by lying down .

Delicious !

Shaken or stirred .

Andre .