Sunday, November 18, 2012

Zinc and Sexiness

Zinc and Sexiness
Andre Willers
18 Nov 2012
Zinc re-absorption via the inner thighs plays a large role in activating homonin sexual pheromones and athletic success .

Discussion :
1.Any chase splatters the inside of the thighs of a cursorial raptor human or canine with urine .
2.The urine contains elements of zinc , which are absorbed through the inner thigh skin and acts as hormones (see Appendix I and III) .
3.This triggers both pheromone production and long-term pheromone receptor site production . Chasing is a cursorial raptor mating ritual .
4.The switch-off : after a successful mating , the positive feedback mechanism (ie sweet taste of Sex) is switched off through zinc-sulfate (see Appendix II) .
5.In other words , over zinc supplements will do horrible things to your sex life and enjoyment of food . The two are related .
Distance athletes routinely pee in their pants . What they don’t realise that this is essential to that “Aha!” endorphine high . It is actually a sexual high .
Of course we can mimic this : a Zinc spray on the inner thighs and NO deoderants should trigger a randy surge to the winning post . (Wifey better be handy.)
Sexual systems are mobilized and extreme violence can occur .
See http// “Post Coital syndrome” Aug 2005

7.The inner thighs of cursorial raptors :
This needs some more examination , especially as regards re-absorption of trace elements acting like hormones .
8. There has been millions of dedicated researchers on this very subject .

In the spirit of dedicated Scientific Inquiry .
Andre .

Appendix I
Zinc and oderant receptors (ie pheromones) are cousins , genetically speaking .
The sequencing of the human genome uncovered that a large number of gene families are often arranged in a clustered organization [1-3]. C2H2 zinc finger (C2H2-ZNF) genes make up ~2% of all the human genes and represent the second largest gene family in humans after the odorant receptor family [4-7].

Appendix II
Zinc affects taste by blocking sweeteners (ie by blocking positive positive feedback mechanisms in the mouth .)
We investigated the ability of zinc sulfate (5, 25, 50 mM) to inhibit the sweetness of 12 chemically diverse sweeteners, which were all intensity matched to 300 mM sucrose [800 mM glucose, 475 mM fructose, 3.25 mM aspartame, 3.5 mM saccharin, 12 mM sodium cyclamate, 14 mM acesulfame-K, 1.04 M sorbitol, 0.629 mM sucralose, 0.375 mM neohesperidin dihydrochalcone (NHDC), 1.5 mM stevioside and 0.0163 mM thaumatin]. Zinc sulfate inhibited the sweetness of most compounds in a concentration dependent manner, peaking with 80% inhibition by 50 mM. Curiously, zinc sulfate never inhibited the sweetness of Na-cyclamate. This suggests that Na-cyclamate may access a sweet taste mechanism that is different from the other sweeteners, which were inhibited uniformly (except thaumatin) at every concentration of zinc sulfate. We hypothesize that this set of compounds either accesses a single receptor or multiple receptors that are inhibited equally by zinc sulfate at each concentration.
Appendix III
There is a lot of Zinc in urine .
Zinc is lost from the body through the kidneys, skin, and intestine. The endogenous intestinal losses can vary from 7 mmol/day (0.5 mg/day) to more than 45 mmol/day (3 mg/day), depending on zinc intake (4). Urinary and skin losses are of the order of 7-10 mmol/day (0.5-0.7 mg/day) each and depend less on normal variations in zinc intake (4). Starvation and muscle catabolism increase zinc losses in urine. Strenuous exercise and elevated ambient temperatures could lead to losses by perspiration.
The body has no zinc stores in the conventional sense. In conditions of bone resorption and tissue catabolism, zinc is released and may be re-utilised to some extent. Human experimental studies with low-zinc diets 2.6-3.6 mg/day (40-55 mmol/day) have shown that circulating zinc levels and activities of zinc-containing enzymes can be maintained within normal range over several months (5, 6), which highlights the efficiency of the zinc homeostasis mechanism. Controlled depletion-repletion studies in humans have shown that changes in the endogenous excretion of zinc through the kidneys, intestine, and skin and changes in absorptive efficiency are how body zinc content is maintained (7-10). The underlying mechanisms are poorly understood.

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