Infinite Clones – Never Age
Andre Willers
17 Apr 2013
Synopsis :
All your cells are but clones of your embryonic cells . The
ability to make error-free clones means an anti-ageing system .
Discussion :
1.See Appendix A and
Appendix C . The secret seems to be in the histone , the disruption of the cell
membrane when new cell-nucleus is implanted and trichostatin .
2. See appendix D . “However, cloned embryos had more acetyl groups on their histones
than normal embryos” . Trichostatin removes acetyl groups on the histone . In
other words , it is an epigenetic factor . And a vital one .
3.Disruption of the cell
wall . See Appendix E . The control systems for the histone systems sits in the
cell-walls . Disrupting them by cutting and implanting a new cell nucleus
disrupts the normal counting apparatus . The bath of trichostatin resets it .
The histones rewind and move back to near the cell wall . The Pucker-up effect
.
4.Nanotech that does
this surgery on every cell seems rather crude .
5. We need a lesion on
every cell on the body , concomitant with a infusion of trichostatin , preferably close to cell-division .
6. Luckily we know
something that might fit the bill : Bavituximab .
“Phosphatidylserine
:
From the
NewScientist article , this is a molecule mostly found sticking out of the
inner lining of the cell-wall . Virus budding entails that this molecule is
then found in particular patterns sticking out of the outside the outer
cell-wall .
We expect
such a mechanism if the cell-wall harbours the meta-controls (ie epigenetics)
of the cell and some multicellular activity (ie virus messenger generation).
Humans have
created antibodies that inactivate or target-for-destruction the bits of the
Phosphatidylserine molecule sticking out of the cell-wall .
This will
break the feedback-cycle of a large range of harmful viruses (apparently
including HIV , Flu's , some cancers) .
Bavituximab
( a name only a Pharma could love) does exactly this . Expect to see a lot of
it .”
7. We can now design a simple
anti-agathic .
Bavituximab plus trichostatin . Dosages to be
determined . Hint : use Avogrado’s number . The number of molecules matter .
8. Well , now we can make an
error-free copy . But we would really like to stabilize on an optimal
physiological age like 26 years .
9. That is why Appendix B is there .
Breast milk from a relative is best , but in a pinch any ethnic relative should
do .
10 . A true anti-agathic (albeit
still simple )
The Recipe :
Bavituximab plus trichostatin
plus breast milk stem cells from a relative .
11. Don’t overdo it . I have
no idea what will happen if you try to go below ages when brain matter gets
reorganised . Does anyone ever want to be a teenager again ?
12. Can this be done
into the genes? I really doubt it . This intervention is Beth(1+) . Trying to
build it into DNA-Epigenetic-Histone structure will run into some very
deep-seated Gaiean feedback systems . You
really don’t want to see an Apoptotic Plague .
13 How green is it ?
Very Green .
Green with envy .
14. A few thousand years
of soapies or scrabbling for food will prepare you for Heaven or Hell , take
your pick . Humans will , off course ,
alternate .
15 . “ No more ducats ,
but plenty of daughters.” The Merchant of Venice after 10 000 years .
Shakespeare would have
loved it .
Andre
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Appendix A
Cloning record broken – next up clones from
faeces
·
11:54 12 March 2013 by Andy Coghlan and Robert Gilhooly
·
For similar stories,
visit the Stem Cells Topic Guide
A mouse has smashed the record for sequential cloning – the
ability to make clones of clones of clones. Now the laboratory behind the feat
will attempt to make clones from cells extracted from mouse fur, stuffed bodies
and excrement.
Teruhiko Wakayama at the RIKEN
Center for Developmental Biology in Kobe, Japan, who carried out the work, says
the technique could help in producing high-quality animals for farms and
conservation purposes. "If a 'super cow' that could produce a lot of milk
or Kobe beef could be cloned at low cost, then not only consumers but also
farmers would be happy," he says.
Twenty-five generations of clones have come from the mouse, and
all 580 of them were healthy, lived normal lifespans and could have healthy
pups through normal mating.
In 2008, Wakayama's team produced clones from dead mice
that had been frozen for 16 years. "My lab is now trying to
make cloned mice from fur, stuffed bodies, and excrement," says Wakayama.
Third-generation clone
Enthusiasm for therapeutic cloning, as the technique that led
to Dolly the sheep is known, waned in the mid-2000s following
scientific fraud scandals in South Korea and the difficulty of producing
animals without abnormalities. Researchers struggled to produce cloned cattle,
pigs, cats and dogs beyond two or three generations.
Now Wakayama's team has emphatically broken
through this barrier thanks to a chemical which more faithfully resets the cell nucleus to
be cloned back to an embryonic state.
First, the team emptied a mouse egg cell of its nucleus. Then they
inserted a nucleus from the adult mouse to be cloned before putting this cell
into a bath of an enzyme blocker called trichostatin. The resulting embryos –
all females – had fewer abnormalities in their histones, the packing materials
for chromosomes.
Previous studies had identified faulty histones in cloned embryos as
a possible reason for the poor success rate of cloning, as well as clone
abnormalities. A possible explanation for the previous limit on the number of
"reclones" is a build up of such abnormalities over successive
generations.
Game changer
"So far, nobody has been able to explain the reason for
this," says Wakayama. "We thought that this limitation was caused by
the accumulation of genetic or epigenetic abnormalities."
"This is very impressive work. If this
translates to other mammalian species – including humans – it could be a major
game changer," says Robert Lanza, chief medical officer at Advanced Cell
Technology, a company based in Marlborough, Massachusetts, that is developing
treatments based on stem cells.
Wakayama says he doesn't know if his technique will make it easier
to clone primates, let alone humans, and has no plans to try. "I am a
mouse researcher and have no experience with other species. I will not even
attempt to use rats, because male rats are extremely difficult to clone."
One application of cloning is to preserve
endangered species, as advocated late last year by Brazil.
The ability to make clones from fur, specimens preserved in museums and
excrement would potentially allow the "resurrection" of extinct
animals.
Journal reference: Cell Stem Cell, DOI: 10.1016/j.stem.2013.01.005
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Appendix B
Are breast milk stem cells the real deal
for medicine?
·
14 March 2013 by Douglas Heaven
·
Magazine issue 2908. Subscribe and save
·
For similar stories,
visit the Stem Cells Topic
Guide
PROTEINS, carbohydrates and vitamins are all on the menu for a
breastfed baby. Now it seems you can add stem cells to that list. Evidence is
piling up that both breast milk and breast tissue contain embryonic-like stem
cells.
That might mean we will soon have access to a source of stem cells
without destroying embryos. This would be a boon as stem cells can turn into
any type of human tissue, making them useful for treating degenerative diseases
like Alzheimer's or for regrowing damaged heart muscle.
In 2011 Foteini Hassiotou at
the University of Western Australia in Crawley and colleagues found stem cells in
lactating breast tissue and breast milk. When they grew the breast
milk cells they turned into the three types of cells from which all tissues and
organs develop – just like human embryonic stem cells (hESCs) do.
Hassiotou has since found this
"pluripotency" in many more breast milk samples and thinks that breast
milk stem cells could one day replace those from embryos. In one study, her
team looked at fresh breast milk from more than 70 healthy breastfeeding women.
They found that BMSCs expressed several genes that are also found in hESCs and
help them replicate. Cultured samples also grew into different tissues
including bone, neuron, heart and pancreatic cells (Human Lactation, DOI: 10.1177/0890334413477242).
In some cases, the team found that 30 per cent of all cells in
breast milk were stem cells. In studies with monkeys and mice the cells were
shown to pass into the bloodstream.
"One can speculate wildly about what they do in the
baby," says Hassiotou. But she thinks that breastfed infants could be
getting a developmental head start, with stem cells from the mother
contributing to organ development in the newborn.
However, BMSCs fail one widely accepted test for embryonic cells:
when injected into mice, they don't form a type of tumour called a teratoma.
For many this failure is a deal-breaker.
But BMSCs are not alone. Mari Dezawa at
Tohuku University in Sendai, Japan, and colleagues have found pluripotent cells
called MUSE cells in bone marrow and connective tissue that do not form
teratomas.
"The best stem cells might not make tumours," says
Hassiotou. Dezawa agrees, and says that MUSE and BMSCs could be superior if
they turn into a wide variety of cells without the risk of forming tumours.
Thea Tlsty at
the University of California in San Francisco says she can imagine that there
are pluripotent cells that do not make tumours. Her team recently identified
pluripotent stem cells in breast tissue from non-lactating women and men (PNAS, doi.org/krn).
Although her cells do form tumours, she agrees that the standard test needs
revisiting.
Tlsty is not convinced that BMSCs are truly pluripotent, since
they have yet to be shown to differentiate fully into living tissue.
Nevertheless, she is open-minded. It used to be thought that these cells were
restricted to the testes or ovaries, "now we're finding them all over the
body".
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Appendix C
Trichostatin
A (TSA) is an organic compound
that serves as an antifungal antibiotic and selectively inhibits the class I and II mammalian histone deacetylase (HDAC)
families of enzymes, but not class III HDACs (i.e., Sirtuins).[1] TSA inhibits the eukaryotic cell cycle during the beginning of the
growth stage. TSA can be used to alter gene expression by interfering with the
removal of acetyl groups from histones (histone deacetylases,
HDAC) and therefore altering the ability of DNA transcription factors to
access the DNA molecules inside chromatin. It is a member of a larger class ofhistone deacetylase
inhibitors (HDIs or HDACIs) that have a broad spectrum of
epigenetic activities. Thus, TSA has some potential as an anti-cancerdrug.[2] One suggested mechanism is that
TSA promotes the expression of apoptosis-related genes,
leading to cancerous cells surviving at lower rates, thus slowing the
progression of cancer.[3] Other mechanisms may include
the activity of HDIs to induce cell differentiation, thus acting to
"mature" some of the de-differentiated cells found in tumors. HDIs
have multiple effects on non-histone effector molecules, so the anti-cancer
mechanisms are truly not understood at this time.
TSA
represses IL
(interleukin)-1β/LPS (lipopolysaccharide)/IFNγ
(interferon γ)-induced nitric oxide synthase (NOS)2
expression in murine macrophage-like cells but increases LPS-stimulated
NOS2 expression in murine N9 and primary rat microglial cells.[5]
However,
there are few if any clinical trials of TSA.[6]
[edit]See also
·
Moon C, Kim SH (June 2009).
"Use of epigenetic modification to induce FOXP3 expression in naïve T
cells". Transplant Proc. 41 (5): 1848–54.doi:10.1016/j.transproceed.2009.02.101. PMID 19545742.
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Appendix D
Glimpse into cloned embryos reveals problems
·
16:46 30 June 2004 by Claire Ainsworth, Berlin
A glimpse of what happens to the DNA in a cell
nucleus during cloning has been revealed by US researchers. They hope their
work will shed light on why cloning creates abnormalities and whether fears
about IVF techniques causing abnormalities in children are justified.
Cloned animals are often born with birth
defects, such as "large offspring syndrome" where the animals are
born much bigger than normal. These defects are linked to problems with
imprinting, the chemical marks on chromosomes that govern the activity of
genes.
Recently, a number of reports have claimed that
the rates of human imprinting disorders are higher in children conceived using
artificial methods, such as IVF and ICSI. One of these disorders,
Beckwith-Wiedemann Syndrome, results in babies being born abnormally large.
Takumi Takeuchi and Gianpiero Palermo from
Cornell University in New York decided to investigate what happens to the DNA
of mouse embryos produced via artificial methods and cloning.
They compared mouse embryos conceived naturally
with ICSI embryos, parthenogenetic embryos - created by stimulating an egg to
divide without being fertilised by sperm - and embryos made by cloning, i.e.
adding an adult cell nucleus to an egg that has had its DNA removed.
Modified behaviour
The team looked at the structure of proteins
called histones in all these embryos. Histones help package DNA into
chromosomes, and also influence the activity of genes. Cells add or remove
different chemical groups to these histones to modify the behaviour of their
genes. Histones in adult cells have very different patterns of chemical groups
to those in embryonic ones.
Takeuchi and his colleagues looked at these
chemical groups with the help of antibodies attached to fluorescent dyes. They
found that the chemical marks on the histones of the nucleus in the cloned
embryos was initially completely re-set, resembling the pattern seen in a
mature egg nucleus.
This means that mature eggs have the ability to
reprogram this aspect of an adult nucleus's behaviour, he told the annual
conference of the European Society for Human Reproduction and Embryology in
Berlin, Germany, on Wednesday.
"We don't know what it means yet,"
Palermo told New Scientist. Clearly, the egg has
taken the nucleus back towards a more immature state, but the extent of the
re-setting is unclear. "Whether it is correctly reprogrammed, we don't
know."
Abnormal activity
Takeuchi also found that at a later stage of
development, the histones of ICSI and parthenogenetic embryos behaved in the
same way as naturally conceived embryos.
However, cloned embryos had more acetyl groups
on their histones than normal embryos, suggesting that the activity of their
genes was abnormal.
This correlated well with the team's finding
that only 30 per cent of the cloned embryos reached a later stage of
development called a blastocyst. The parthenogenetic and ICSI embryos achieved
that stage at a similar rate to naturally conceived embryos.
"We found significantly impaired
development in the cloned embryos compared with those derived from more
conventional assisted reproductive technologies," said Takeuchi. "And
this has made us more convinced that reproductive cloning is unsafe and should
not be applied to humans."
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Appendix E
Phene Systems II
Andre
Willers
4 Feb 2011
Synopsis :
Cellular
meta-control mechanisms (Phenes) situated on the cell- and nucleus wall are
expanded on in more depth .
Discussion :
The article
"The Inner life of the Genome" by Tom Miseli in the Feb 2011 Scientific
American (p46) pulled together a number threads previously discussed in this
blog .
Briefly ,
he found that DNA is organized in the following physical arrangements in a
working cell-nucleus :
DNA ->
Wound around Histone Spools -> Histone Spools fold to form Chromatin strings
-> Heterochromatin strings (a very tightly folded Chromatin strings) (see
para 6 below)
Heterochromatin
is so tightly wound that the DNA is inaccessible to transcription factors (ie
inert , switched off) . They are found mostly near the nucleus wall . The
Chromatin strings form delineated tangles , with preferred positions in the
nucleus space .
Near the
center are volumes called Transcription factories .
This is
where the rubber hits the tar .
Volumes
rich in aggregations of cellular components , polymerase enzymes and
transcription factors . How did they get there ? See point 4 below about
Chaperones and Self-organization .
Genes on
the outer Heterochromatin string , on activation from the Phene system ,
unwinds to make the DNA accessible . The chromatin string simultaneously also
migrates inward to a transcription factory (presumably triggered by the phene
signal) and gets expressed .
This has
been described as the Histone Code forming the epigenetic system
From evolutionary considerations ,
meta-control systems on cell-level and nucleus level are taken to be related .
Not only
are they descended from the same ur-mechanisms , but if they were not related ,
that would add another expensive translation layer between cell-wall and
nucleus-wall , with concomitant chances of error .
Evolutionary
pressures would have elided any such mutation .
(Sounds
logical , but is it true ? I suspect it is usually true , but exceptions will
be found .
The
perversity of animate matter exceeds that of inanimate matter by orders of
magnitude)
The Histone System can then be seen as an
important subset of the Epigenetic System
But the
epigenetic system can have signals that are not relevant to cells .
See
"Tunneling nanotubes"
See
the following main threads :
http://andreswhy.blogspot.com
"Rife and the Histone Code" Jan 2011
http://andreswhy.blogspot.com
"Phene System" Nov 2009 (reproduced in Appendix A below for
convenience .)
http://andreswhy.blogspot.com
"ATP as a neurotransmitter" Jan 2010
http://andreswhy.blogspot.com
"Tunneling nanotubes" Nov 2010
http://andreswhy.blogspot.com
"Coffee foam" Aug 2009
http://andreswhy.blogspot.com
"Intralipid II" Feb 2011
http://andreswhy.blogspot.com
"Chaperones , Unpacking and Asthma" Aug 2008
Points to note :
1;
"Distributed viruses" as theorized in "ATP as a
Neurotransmitter" can be better described as pleomorphic organisms as set
out in "Rife and the Histone code"
2;Intralipid
is important because of its ability to hide epigenetic triggers . Notice the
importance of phosphatydilserine (see "Phene Systems") . What would
be the effect of phosphatydilserine with
Intralipid-type mechanisms ?
3;
Switching off genes near the cell-wall is a typical evolutionary fail-safe
control .
(A process
is inhibited , unless that inhibition is inhibited in turn)
4; When a
gene in the outer-tangle of Heterochromatin strings is activated , the loop it
is in drops in nearer to a Transcription factory .
How does it
know where to go ?
One
theorizes that initially it is self-organizing (ie a hit-or-miss affair) . But
various Beth levels will force an evolution of chaperones (see http://andreswhy.blogspot.com
"Chaperones , Unpacking and Asthma" Aug 2008 )
So , one
envisages a mixture of chaperones(2/3) and self-organization (1/3) (http://andreswhy.blogspot.com
"NewTools reserves" Nov 2008 . Error arguments.)
5;
Chromosome abnormalities (like cancer) . Without a chaperone the dangling loop
is vulnerable to breaking and then joining up with an unsuitable partner . Like
in any bad marriage , the children suffer .
6.Stem Cells :
The above
description is for a mature , functioning differentiated cell .
Things are
different just before and after divisions .
In
Embryonic stem-cells , there are no Heterochromatin strings .
All the
genes are active , but the shebang is not very robust .
On
receiving a phene-signal to differentiate the cell , lamin proteins are formed
and fold Chromatin strings -> Heterochromatin strings and also tether them
to the nucleus wall . The nucleus becomes much more robust .
Lamin
proteins can then be classified as chaperones .
But notice
how easily nearly any cell can be turned back into a pluripotent cell .
The body
does this all the time (lately , stem cells have been created out of cells
found in urine .This is very unlikely
for a storage mechanism .)
From
Wikipedia :
"The nuclear lamina consists
of a two-dimensional matrix of proteins located next to the inner
nuclear membrane. Thelamin family of
proteins make up the matrix and are highly conserved in evolution. During mitosis, the lamina matrix is reversibly
disassembled as the lamin proteins are phosphorylated. Lamin proteins are thought to
be involved in nuclear stability, chromatin structure
and gene expression."
The lamins are thriftily
reprocessed during mitosis . This means there is an programmed biological
pathway to reverse differentiation (ie create stem cells) . Which why stem
cells are so easily created .
The only problem now is why the
body does not do it more frequently (ie why grow old?) It seems that the phene
system has a memory , and the new cells after mitosis are reprogrammed .
Any helpful suggestions are
welcome .
(Old age sucks.)
7; Heat pulses and Timing .
From para 4
above (Self-organization) , random movement due to heat promotes
self-organization .
The
molecules creating timing mechanisms are evolutionary very old (recent
findings) . Heat pulses at harmonics of the diurnal rhythm should entrain the
self-organizing process and reduce copying errors quite drastically .
(Remember ,
a modest 10 degree Celsius rise in temperature doubles chemical activity on a
molecular level)
The Tea Ceremony .
An existing
and proven technology .
The number
of seconds in a day = 3^3 * 2^7 * 5^2 = 86 400
Sipping a
very hot liquid every x seconds will pulse with resonances at
X=
2,3,4,5,6, 8,9,10,12.15,16,18,20,24, 25,27,30,… seconds
About 2-3
seconds per sip seems doable . The liquid must be kept hot and the sip rate
maintained . This is ok for stomach systems .
We know
long-term nerve potentiating is about 10 minutes , so if nervous system is
targeted , sip slower (but not longer than 6 seconds between sips) and the tea
must be kept at the same temperature . Eg for 6 seconds between 2.5 ml sip ,
you will drink 10*60/6*2.5 = 250 ml (about one cup .)
Will a Tea
Ceremony with iced tea have the same effect ?
If the
nervous system is targeted , yes . Entrainment is the goal . So the bigger the
temperature difference , more the body will notice . Remember the caffeine .
The maximum
effect if you want to target the nervous system (relax, etc) would be then to
do the Tea Ceremony and make each alternate sip iced tea and very hot tea as
described above . Each in a demi-tasse .
I'm afraid
Tea Ceremony purists in Japan and China will not welcome this conclusion
In other
cases , you can experiment , but I estimate that hot alone will work .
Bar flies ,
hot chocolate and hot/iced coffee I leave for enthusiastic students .
Or else you
can pulse infrared at these rates and frequencies derived from Rife's work
Or else sit
in the sun with a shade that fluctuates (old style Eastern potentate , or
rotating sun-umbrella with variable panels)
Or else
watch TV . Heat radiation on old cathode-ray TV's will resonate at 25 , 27 ,30
times per second .
Children
sitting close will be more susceptible .
Watching TV
on CRT's makes them healthier ? Some
independent verification is required here .
Computer
screens and games ditto .
That was
fun !
Andre
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Appendix A
Phene Systems.
Andre
Willers
5 Nov 2009
Synopsis :
Cellular-wall
control systems(phenes) of epigenetic and virus systems are becoming known and
described .
Discussion :
See
NewScientist 8 Aug 2009 p 41 "Kills all known germs" by B Holmes .
Relevant
research by Philip Thorpe at University of Texas Southwestern Medical Center in
Dallas .
See
also http://andreswhy.blogspot.com
"Coffee Foam" 2 Aug 2009 and later .
The argument is simple : self-replicating structures
started on the walls of bubbles (probably clay : Gecko life) . These developed
into the present day epigenetic control structures of processes inside the
bubbles (ie cells) .
Viruses are
messengers/controls both for intra-cellular and extra-cellular use .
Which is
why they are usually wrapped in variants of the cell-wall .
Phosphatidylserine :
From the
NewScientist article , this is a molecule mostly found sticking out of the
inner lining of the cell-wall . Virus budding entails that this molecule is
then found in particular patterns sticking out of the outside the outer
cell-wall .
We expect
such a mechanism if the cell-wall harbours the meta-controls (ie epigenetics)
of the cell and some multicellular activity (ie virus messenger generation).
Humans have
created antibodies that inactivate or target-for-destruction the bits of the
Phosphatidylserine molecule sticking out of the cell-wall .
This will
break the feedback-cycle of a large range of harmful viruses (apparently
including HIV , Flu's , some cancers) .
Bavituximab
( a name only a Pharma could love) does exactly this . Expect to see a lot of
it .
The question is :
Why are
these antibodies not part of the body's normal immune system ?
Answer:
The virus
system is also used for information carrying .
Suppressing
the virus system completely will destroy a multicellular organism , and make it
revert to a collection of individual cells , even down to elemental chemicals
if done properly . (Cf Ebola , SuperEbola ,
etc)
This can be
bypassed by shining intense , quantum-entangled infra-red light through the
mess (see previous posts) .
The normal
system has to play a delicate balancing act between destruction from bad
viruses and good,communication viruses .
If the
baddies get too big an influence , intervention might be required . This is the
duty of the epigenetic system . Why is it falling down on the job ?
The Dendritic Immune sytem
This is the
connection between the synapse-type learning systems and the phene-system in
the cell walls .
The
dendritic immune system's effectiveness
is magnified by at least 3 orders of magnitude by a continuous sharp
pressure-differential . The search space is drastically reduced by stochastic
resonance . (The proof is either immediately obvious or very long).
Hence
modern music and it's prevalence .
Your
chances of survival is about 10^3 better if you listen to some semi-random
rock.
Hence iPod and
others . This is a real effect .
But we can
do better than that by internal Click programming .
You know :
Clicking the tongue , beak , teeth , exoskelet .
There is no
animal with synapses or an immune system that makes no clicks at some stage .
This is
actually quite amazing .
Plants:
We would
then expect plants to be noisy . And they are , on very low frequencies . This
is how desert elephants survive : they hear the bulbous plants growing by the
low-frequency pulses sensed through their feet . A patch of bulbs would have a
signature low-frequency pulse observable from a long way off . (The bulbs would
evolve-learn to spurt growth at the same time . This would lessen the chance of
detection and eating by close-by herbivores not as smart as elephants)
Click-talking plants .
Better than
just talking to them . But try some castanets . Tap-dancing will work too , but
the neighbours will think you're really crazy tap-dancing to the plants .
Tojours .
Grinding your teeth .
A common
complaint , usually explained away as stress . Actually , your teeth are trying
to click together . A part of the body's housekeeping routine . Programming the
gums and intestinal tract .
Just for
the hell of it , click your teeth together 5 times together with 5 double-tongue
clicks interspersed as you feel . Count them . You will feel an immediate
stress-relief feeling . Blood pressure will drop . Be careful if medication to
lower blood pressure is taken .
I just
thought of the above , and tried it .
The teeth
(molars mostly) must click against each other . You will notice there is a
damage-control protocol hardwired in . The teeth will click , but not hard
enough to cause damage . This already tells us that this is an old system .
The natural
tendency will be for a double-teeth click , followed by a double tongue-click
with the mouth open . The process then repeats , varying the teeth , tongue and
mouth open/closed .
Be careful
of hypotension of the cardiovascular system . The stress relief is very
pronounced . Especially around the neck and upper shoulder muscles .
Teeth-grinding
should cease .
But why ?
I did not
expect this . But the effect is so pronounced that I cannot ignore it .
The neck
and shoulder muscles do not relax through
an effort of will or exercise . They just relax . (Sort of melt) .
Maybe a
double bite-bite on empty air signals the end of an aggressive episode , and
for expensive fight-or-flight mechanisms to stand down .
This would
make this fairly deep-wired , certainly deeper than psychological stress .
Phene effects .
The
stand-down is an immediate stand-down , regardless of the number previous
stress-generating events . This must be true , since this is the epigenetic
programming system . There is no other memory system .
Phene
programming of immune-systems , bone-growth , digestion , neural growth etc
,are immediately affected . Genes are switched from crisis to maintenance .
Using the carbohydrate-energy mechanism is a short-term crisis mechanism .
Standing
down the mechanism should ameliorate conditions like Diabetes II .
Or getting
fat .
Want to get thin ?
The
algorithm :
Double-teeth
click , followed by a double tongue-click with the mouth open . The process
then repeats , varying the teeth , tongue and mouth open/closed .
Repeat at least ten times a month .
That’s it .
Can it be this simple?
Yes .
Why has nobody thought of it before ?
As far as I
am aware , nobody in our recorded history has thought that the click of teeth
together can have physiological effects . Prehistoric societies were probably
aware of the effect .
Yet it does
, as you can find out for yourself .
The same
for the simplicity . Simple causes has wide effects , because they are so basic
. You must look at the whole argument .
In any case
, there is hardly any risk .
I am doing
it . Tojours!
Hasta la
vista !
Coming or
going .
Andre .
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